Clinics

Man is the only animal that is infected by this virus. There are five different types of viral hepatitis infection , A,B,C,D & E. A & E are spread through contaminated water and food. B & C through blood and semen . 20% of the people suffering from serious liver ailments have Hepatitis E virus as its cause and Hepatitis D virus has an ability to catalyses the infection from other types and make the patient reach an advanced stage in a short time. Annually Hepatitis B is the cause for the death in about 2 million people across the world. A patient infected by Hepatitis B virus will mostly die of liver cirrhosis or liver cancer. In patients, that have a weak immune system B- virus starts as a carrier and ultimately the patient dies as a victim of Hepatitis-B. If a person has a strong immune system then he can withstand the attack of the virus and live a normal healthy life. Hepatitis –B is seen in two different ways in patients 1. Liver enlargement with jaundice 2. Asymptomatic but infected in the blood. In the first case, being symptomatic, it makes the treatment easier. In the second case since it is asymptomatic , treatment is difficult. In 1976, Dr. V.S. Blumberg received the nobel prize for discovering Hepatitis-B virus . Only after this did we have knowledge about it. This disease has been prevalent in the world much prior to this discovery. Yet the clear picture came into being only after the discovery. Statistically more than 40 crores of people across the world have the disease.Total number of infected people worldwide is 200 crores. This disease is most commonly seen in third world countries . After China , India has the highest percentage of infected people. PERCENTAGE PREVALENCE OF HBV WORLDWIDE 0 –1 % : North America, Western Europe, Australia 2 – 5% : South east Europe, Middle east countries, West Asia, Indian Sub continent , South Central America 5% & above : Africa, East Asia Pacific , Asia minor , Caribbean islands. In developed countries , the number of Hepatitis –B cases are less , but the maximum cases are seen in the younger people. At the same time , in underdeveloped countries , most of the cases are children. This has made the seriousness of the matter worse, as 90% of the infected children are unable to save themselves from the disease. Whereas in youngsters, 90% of the patients can be saved. In developed countries , there is a continuous drop in the percentage of people infected with Hepatitis-B. In comparison to this , in underdeveloped countries , including India, there is a continuous increase in the percentage. As per the survey , the present percentage of carriers is 4.5 to 5% of the total population. That means 4.5 to 5 crore Indians are infected by HBV. One in two cases of chronic liver disease and eight in ten cases of primary liver cancer is due to Hepatitis –B infection. It is recorded that liver disease due to Hepatitis –B infection is considered to be the fourth or fifth important cause of death in an individual’s most productive period of time [ 15 to 45 years] . Even in a small state like Kerala, the percentage of people infected by HBV is not at all far behind the national level, i.e 5% [ Carriers]. 1.How does one get infected by Hepatitis- B virus ? Man is the only known reservoir of this virus . It is transmitted through infected blood and other body fluids such as semen, vaginal secretion and wound exudates. There are 4 major routes of transmission :  Parenteral/ percutaneous : During transfusion of infected blood and through infected needles.  Horizontal : Between person to person ( accidental contact with infected blood and other body fluids)  Vertical : From infected mother to child at birth.  Sexual : Between sexual partners Accidental contact with such carriers who are unaware about the virus they are harboring, can transmit the virus to others. 2. What are the symptoms of Hepatitis-B infection ? The majority of infants and children infected with Hepatitis –B do not show any symptoms of Hepatitis-B infection. In the case of adults, a small number of individuals may not show any symptoms at all. The others initially developed flu like symptoms such as loss of appetite, tiredness , chills , mild fever, headache, body ache , vomiting and internal pain on the side of the location of the liver. Later jaundice or jaundice like symptoms [ pale faeces , dark urine, and yellowness in the eye ] may develop. In youngsters vomiting and pain will be comparatively more in the beginning, but after developing jaundice, they disappear . Their eyes, skin, urine and the skin under the tongue develops yellowness. In most of the patients, the spleen gets enlarged , the liver shrinks and fluid accumulates in the stomach. These symptoms indicate the chronic condition of the disease. Faeces will be either whitish or blackish and there will be itching all over the body. The disease can be detected by testing the blood. 3. What is an HBV carrier ? An HBV carrier has no symptoms of the disease, but when tested is found to be HbsAg Positive. 4. Prevention of spreading of Hepatitis 1. Check blood used for treatment. 2. Sterilise all medical instruments like syringes etc. 3. Do not have more than one sexual partner. 4. Take Kamilari [ a medicine manufactured by M/S. NUPAL , COCHIN – 20 , for viral Hepatitis ] 2 caps 2 times daily for one month every year , this will ensure prevention . 4.Who are at high risk ? 1. At high risk due to profession All medical and para medical personnel such as doctors, nurses and people working in pathology labs, blood banks, dialysis units and cancer units because their profession exposes them to the risk of dealing with blood and body fluids of patients who may be carriers. 2. At high risk due to illness. a. Patients like thalassemics and haemophilics who receive blood or blood related products. b. Patients on dialysis. 3. At high risk due to sexual or social habits a. Heterosexuals with multiple sexual partners, b. Homosexuals and prostitutes c. People who play contact sports d. People who undergo tattooing We have effective treatment for Hepatitis B& C. Patient has to take the following blood tests before starting the course of treatment and send us the reports. HbsAg [by CMIA] Quantitative AntiHbsAg [ by CMIA] & LFT.

Normal platelet counts range from 150,000 to 400,000 per cu/ml. Those with ITP have a lower platelet count. It can range from severe cases that hover close to zero to more mild cases where the counts stay in closer to 100,000. 30,000 is often considered a ‘safe’ count’, one that is high enough to protect against cerebral hemorrhage.
In people with ITP the platelets are often enlarged. They stay in the blood stream from a few hours to close to the normal eight to ten days depending on the severity of the disease.
Platelets play a crucial part in the blood clotting process by forming a platelet plug. This is a two step process. First, single platelets bind to the site of the wound (adhesion). Next, the platelets bind to each other (activation). Activation can be stimulated by components released when the blood vessel is damaged and by thrombin, released during the blood clotting process. When platelets become activated they change. They release agents which recruit and activate the surrounding platelets. The result of these two processes is the formation of fibrin which stabilizes the platelet plug, stops bleeding and allows injuries to heal.
In your body, 2% of the serotonin, a mood elevating neurotransmitter, is stored in platelets. In addition to serotonin, your platelets also carry its ‘parent’ or precursory chemical L-tryptophan. While serotonin can’t pass through the blood brain barrier, L-tryptophan can. These substances are involved in such processes as sleep/wake cycles, biological rhythms, appetite, mood regulation, etc.
We have effective treatment for increasing platelet count . Many patients after taking various treatments have found our treatment to be the best.
We have been treating outdoor patients all over the world for decades from our Speciality clinic at Cochin and through mail order.

Please send us your investigation reports along with your case history by e-mail . We will accordingly send you the details of our treatment along with the cost of treatment.

Stomach ulcer (also called a peptic ulcer) is small erosion (hole) in the gastrointestinal tract. The most common type, duodenal, occurs in the first 12 inches of small intestine beyond the stomach. Ulcers that form in the stomach are called gastric ulcers. An ulcer is not contagious or cancerous. Duodenal ulcers are almost always benign, while stomach ulcers may become malignant.

The size of a stomach ulcer can range between 1/8 of an inch to 3/4 of an inch.

Children develop stomach ulcers too.Stomach Ulcers may be a symptom of another disease or condition. Ulcers are often common in mastocytosis. Bleeding from stomach ulcers may cause iron deficiency anemia.

What causes stomach ulcers?

The direct cause of peptic ulcers is the destruction of the gastric or intestinal mucosal lining of the stomach by hydrochloric acid, an acid normally present in the digestive juices of the stomach. Infection with the bacterium Helicobacter pylori is thought to play an important role in causing both gastric and duodenal ulcers. Helicobacter pylori may be transmitted from person to person through contaminated food and water. Antibiotics are the most effective treatment for Helicobacter pylori peptic ulcers.

Injury of the gastric mucosal lining and weakening of the mucous defenses are also responsible for gastric ulcers. Excess secretion of hydrochloric acid, genetic predisposition, and psychological stress are important contributing factors in the formation and worsening of duodenal ulcers.

Another major cause of ulcers is the chronic use of anti-inflammatory medications, such as aspirin. Cigarette smoking is also an important cause of ulcer formation and ulcer treatment failure.

Symptoms of stomach ulcers

The major symptom of an ulcer is a burning or gnawing feeling in the stomach area that lasts between 30 minutes and 3 hours. This pain is often interpreted as heartburn, indigestion or hunger. The pain usually occurs in the upper abdomen, but sometimes it may occur below the breastbone. In some individuals the pain occurs immediately after eating. In other individuals, the pain may not occur until hours after eating. The pain frequently awakens the person at night. Weeks of pain may be followed by weeks of not having pain. Pain can be relieved by drinking milk, eating, resting, or taking antacids.

Appetite and weight loss are other symptoms. Persons with duodenal ulcers may experience weight gain because the persons eat more to ease discomfort. Recurrent vomiting, blood in the stool and anemia are other symptoms.

What does a stomach ulcers affect ?

The main thing that a stomach ulcer affects is the nerves surrounding it. The nerves become agitated and cause a great amount of pain. However, stomach ulcers can cause hemorrhages from the erosion of a major blood vessel; a tear in the wall of the stomach or intestine, with resultant peritonitis; or obstruction of the gastrointestinal tract because of spasm or swelling in the area of the ulcer.

We have effective treatment for peptic ulcers, we are able to give immediate relief to the patients and gradually over a period of time, completely cure him. We have a strike rate of more than 80% of patients treated for this indication.

We have been treating outdoor patients all over the world for decades from our Specialty clinic at Cochin and through mail order.

Please send us your investigation reports along with your case history by e-mail. We will accordingly send you the details of our treatment along with the cost of treatment.

Renal stones are a common occurrence, being present at some time in one in ten of the population, although a significant proportion will remain asymptomatic. Men are more commonly affected than women with a male to female ratio of 3:1.The peak age for developing stones is between 30 and 50, and recurrence is common.

Risk Factors Several risk factors are recognised to increase the potential of a susceptible

individual to develop stones, these include:

Anatomical anomalies in the kidneys and/or urinary tract e.g. horseshoe kidney, ureteral stricture
Family history of renal stones
Hypertension
Gout
Hyperparathyroidism
Immobilisation
Relative dehydration
Metabolic disorders which increase excretion of solutes e.g. chronic metabolic acidosis, hypercalciuria , hyperuricosuria etc.
Climate, more common in hot climates.

Renal calculi are developed from crystals which form when the urine is supersaturated with salt and minerals such as calcium oxalate, struvite, uric acid and cystine.1They vary considerably in size from small ” gravel like” stones, to large ” staghorn” calculi. The calculi may stay in the position in which they are formed, or may migrate down the urinary tract producing symptoms along the way. Recent studies suggest that the initial factor involved in the formation of a stone may be the presence of nanobacteria that form a calcium phosphate shell. These small intracellular bacteria are present at the centre of over 95% of all stones.

Presentation
Symptoms Many stones will cause no symptoms at all, and will merely be discovered during investigations for other conditions. Other stones may be diagnosed by a classical presentation of colicky abdominal pain radiating from the loin to the groin. Other symptoms which may be present include:-

Rigors and fever
Dysuria
Hematuria
Urinary retention
Nausea and vomiting.

Take a detailed history including family history.

Examination: especially for palpable kidneys and bladder.

Differential Diagnosis Other diagnoses which must be considered in the acute situation are other causes of colicky abdominal pain e.g. bowel obstruction, strangulated hernia, pain due to bowel muscle spasm, appendicitis ,other forms of renal colic such as “clot” colic secondary to trauma and bleeding of the kidney and other causes of urinary retention such as prostatism.

We have effective treatment for Renal stone , we are able to break the stones into smaller stones and are expelled through urine. The period of treatment varies from a few weeks to a few months ,depending on the seriousness of the case.

HIV begins its infection of a susceptible host cell by binding to the CD4 receptor on the host cell. CD4 is present on the surface of many lymphocytes, which are a critical part of the body’s immune system. Recent evidence indicates that a coreceptor is needed for HIV to enter the cell. This recognition of HIV coreceptors and progress in understanding how HIV fuses with the cell has opened up new possibilities for antiviral drugs. A number of new agents are being designed to prevent infection by blocking fusion of HIV with its host cell.

Following fusion of the virus with the host cell, HIV enters the cell. The genetic material of the virus, which is RNA, is released and undergoes reverse transcription into DNA. An enzyme in HIV called reverse transcriptase is necessary to catalyze this conversion of viral RNA into DNA. Inhibitors of reverse transcriptase, such as AZT, were the first anti-HIV medications, and are still a critical part of treating patients who have HIV. Reverse transcriptase inhibitors are divided into two classes-nucleoside analogues and non-nucleoside reverse transcriptase inhibitors-based on their structure and how they inhibit reverse transcriptase.

Once the genetic material of HIV has been changed into DNA, this viral DNA enters the host cell nucleus where it can be integrated into the genetic material of the cell. The enzyme integrase catalyzes this process, and inhibitors of integrase are under study as a new way to block HIV replication. Once the viral DNA is integrated into the genetic material of the host, it is possible that HIV may persist in a latent state for many years. This ability of HIV to persist in certain latently infected cells is the major barrier to eradication or cure of HIV. For this reason, based on our current knowledge, patients must remain on anti-viral therapy for life.

Activation of the host cells results in the transcription of viral DNA into messenger RNA (mRNA), which is then translated into viral proteins. The new viral RNA forms the genetic material of the next generation of viruses. The viral RNA and viral proteins assemble at the cell membrane into a new virus. Amongst the viral proteins is HIV protease, which is required to process other HIV proteins into their functional forms. Protease inhibitors, one of the most potent types of anti-viral medications, act by blocking this critical maturation step. Following assembly at the cell surface, the virus then buds forth from the cell and is released to infect another cell.

Unless the HIV lifecycle is interrupted by treatment, the virus infection spreads throughout the body and results in the destruction of the body’s immune system. With current anti-viral medications, such as reverse transcriptase inhibitors and protease inhibitors, HIV infection can be contained. However, a great deal more must be achieved before AIDS epidemic is brought under control. One important immediate goal is to design new, more potent medications that are easier to take and have fewer side effects. However, the ultimate challenges are to use our understanding of the HIV lifecycle to develop medications that will eradicate HIV from people who are already infected and to create a vaccine that will prevent new infections in the future.

We have palliative treatment for HIV infection , we are able to stop opportunistic illnesses ,increase the weight of the patient and reduce the viral load to a great extent.. Most importantly , our medicine allow the HIV infected patient to lead a normal active life.

We have been treating outdoor patients all over the world for decades from our Speciality clinic at Cochin and through mail order.

Please send us your investigation reports along with your case history by e-mail . We will accordingly send you the details of our treatment along with the cost of treatment.

For optimum cholesterol and triglyceride levels :

What Are Healthy Levels of Cholesterol?

Your total blood cholesterol level

Your total blood cholesterol will fall into one of these categories:

Desirable – Less than 200 mg/dL
Borderline high risk – 200–239 mg/dL
High risk — 240 mg/dL and over

Here is some more explanation about each of these categories.

Desirable

If your total cholesterol is less than 200 mg/dL, your heart attack risk is relatively low, unless you have other risk factors. Even with a low risk, it’s still smart to eat foods low in saturated fat, trans fat and cholesterol, and also get plenty of physical activity. Have your cholesterol levels measured every five years -or more often if you’re a man over 45 or a woman over 55.

Borderline high risk

People whose cholesterol level is from 200 to 239 mg/dL are borderline high risk. About a third of American adults are in this (borderline) group; almost half of adults have total cholesterol levels below 200 mg/dL.

Have your cholesterol and HDL rechecked in one to two years if:

Your total cholesterol is in this range.
Your HDL is less than 40 mg/dL.
You don’t have other risk factors for heart disease.

You should also lower your intake of foods high in saturated fat and cholesterol to reduce your blood cholesterol level to below 200 mg/dL. Your doctor may order another blood test to measure your LDL cholesterol. Ask your doctor to discuss your LDL cholesterol with you. Even if your total cholesterol is between 200 and 239 mg/dL, you may not be at high risk for a heart attack. Some people -such as women before menopause and young, active men who have no other risk factors- may have high HDL cholesterol and desirable LDL levels. Ask your doctor to interpret your results. Everyone’s case is different.

High risk
If your total cholesterol level is 240 or more, it’s definitely high. Your risk of heart attack and stroke is greater. In general, people who have a total cholesterol level of 240 mg/dL have twice the risk of coronary heart disease as people whose cholesterol level is 200 mg/dL.

You need more tests. Ask your doctor for advice. About 20 percent of the U.S. population has high blood cholesterol levels.

Your LDL cholesterol level
Your LDL cholesterol level greatly affects your risk of heart attack and stroke. The lower your LDL cholesterol, the lower your risk. In fact, it’s a better gauge of risk than total blood cholesterol. Your LDL cholesterol will fall into one of these categories:

LDL Cholesterol Levels

Less than 100 mg/dL – Optimal
100 to 129 mg/dL- Near Optimal/ Above Optimal
130 to 159 mg/dL – Borderline High
160 to 189 mg/dL – High
190 mg/dL and above – Very High

The key point to remember is, the lower your LDL cholesterol, the lower your risk. Your doctor may prescribe a diet low in saturated fat and cholesterol, regular exercise and a weight management program if you’re overweight. If you can’t lower your cholesterol with these efforts, medications may also be prescribed to lower your LDL cholesterol. Check these categories and the goals for treatment that can lower your risk of heart attack.

Our treatment for imbalanced Cholesterol is very effective and we are able to bring the HDL , LDL and triglyceride level within acceptable limits, hence reducing the coronory index..

We have been treating outdoor patients all over the world for decades from our Speciality clinic at Cochin and through mail order.

Please send us your investigation reports along with your case history by e-mail . We will accordingly send you the details of our treatment along with the cost of treatment.

Rheumatoid arthritis is an autoimmune disease that causes chronic inflammation of the joints. Rheumatoid arthritis can also cause inflammation of the tissue around the joints, as well as other organs in the body. Autoimmune diseases are illnesses that occur when the body tissues are mistakenly attacked by its own immune system. The immune system is a complex organization of cells and antibodies designed normally to “seek and destroy” invaders of the body, particularly infections. Patients with autoimmune diseases have antibodies in their blood that target their own body tissues, where they can be associated with inflammation. Because it can affect multiple other organs of the body, rheumatoid arthritis is referred to as a systemic illness and is sometimes called rheumatoid disease.

While rheumatoid arthritis is a chronic illness, meaning it can last for years, patients may experience long periods without symptoms. Typically, however, rheumatoid arthritis is a progressive illness that has the potential to cause joint destruction and functional disability.

A joint is where two bones meet to allow movement of body parts. Arthritis means joint inflammation. The joint inflammation of rheumatoid arthritis causes swelling, pain, stiffness, and redness in the joints. The inflammation of rheumatoid disease can also occur in tissues around the joints, such as the tendons, ligaments, and muscles.

In some patients with rheumatoid arthritis, chronic inflammation leads to the destruction of the cartilage, bone and ligaments causing deformity of the joints. Damage to the joints can occur early in the disease and be progressive. Moreover, studies have shown that the progressive damage to the joints does not necessarily correlate with the degree of pain, stiffness, or swelling present in the joints.

The disease is three times more common in women as in men. It afflicts people of all races equally. The disease can begin at any age, but most often starts after age forty and before sixty. In some families, multiple members can be affected, suggesting a genetic basis for the disorder.

Most effective treatment for Rheumatoid Arthritis is only available through ayurvedic treatment . We have effective treatment that brings the R-factor down to the normal level and allows the patient to live normal healthy life .

We have been treating outdoor patients all over the world for decades from our Speciality clinic at Cochin and through mail order.

Please send us your investigation reports along with your case history by e-mail . We will accordingly send you the details of our treatment along with the cost of treatment.

Osteoarthritis is a type of arthritis that is caused by the breakdown and eventual loss of the cartilage of one or more joints. Cartilage is a protein substance that serves as a “cushion” between the bones of the joints. Osteoarthritis is also known as degenerative arthritis. Among the over 100 different types of arthritis conditions, Osteoarthritis occurs more frequently as we age. Before age 45, osteoarthritis occurs more frequently in males. After age 55 years, it occurs more frequently in females.

We have palliative treatment for Osteo arthritis, wherein we are able to delay further damage to the cartilage. Patients treated by us have reported good relief and ability to live a normal active life.

Psoriasis is an inflammatory skin condition. There are five types, each with unique signs and symptoms. Between 10% and 30% of people who develop psoriasis get a related form of arthritis called “psoriatic arthritis,”which causes inflammation of the joints.Plaque psoriasis is the most common type of psoriasis. About 80% of people who develop psoriasis have plaque psoriasis, which appears as patches of raised, reddish skin covered by silvery-white scale. These patches, or plaques, frequently form on the elbows, knees, lower back, and scalp.However, the plaques can occur anywhere on the body.

The other types are guttate psoriasis (small, red spots on the skin), pustular psoriasis
(white pustules surrounded by red skin), inverse psoriasis (smooth, red lesions form in skin folds), and erythrodermic psoriasis (widespread redness, severe itching, and pain).

Regardless of type, psoriasis usually causes discomfort. The skin often itches, and it
may crack and bleed. In severe cases,the itching and discomfort may keep a person awake at night, and the pain can make everyday tasks difficult.

Psoriasis is a chronic, meaning lifelong, condition because there is currently no cure.
People often experience flares and remissions throughout their life. Controlling the signs and symptoms typically requires lifelong therapy.

Treatment depends on the severity and type of psoriasis. We are able to cure 70% of patients referred to us.

We have been treating outdoor patients all over the world for decades from our Speciality clinic at Cochin and through mail order.

Please send us your investigation reports along with your case history by e-mail.We will accordingly send you the details of our treatment along with the cost of treatment.

An anal fissure is a small tear or cut in the skin lining the anus which can cause pain and/or bleeding.

What are the symptoms of anal fissure?

The typical symptoms of an anal fissure are extreme pain during defecation and red blood streaking the stool. Patients may try to avoid defecation because of the pain.

What are the causes of anal fissure?

A hard, dry bowel movement can cause a tear in the anal lining, resulting in a fissure. Other causes of a fissure include diarrhea and inflammation of the anorectal area.

Anal fissures may be acute (recent onset) or chronic (present for a long time or recurring frequently). An acute fissure is usually due to altered bowel habits while a chronic fissure may be either due to poor bowel habits, overly tight or spastic anal sphincter muscles, scarring or an underlying medical problem

We have effective treatment for Fissure patients , we are able to fully cure the patients over a period of time with our oral ayurvedic medicines ,that is without any surgery.

We have been treating outdoor patients all over the world for decades from our Speciality clinic at Cochin and through mail order.

Please send us your investigation reports along with your case history by e-mail . We will accordingly send you the details of our treatment along with the cost of treatment.